The findings point toward potential new avenues for treating post-traumatic stress disorder (PTSD).
Key Facts:
- The researchers successfully controlled fear responses in mice by increasing brain plasticity and inducing desensitization to fear memories.
- The process involved decreasing the expression of the Acan gene, responsible for the production of the protein Aggrecan, which reduces brain malleability.
- This research could pave the way for improved treatment options for adults with PTSD, potentially enhancing the success of exposure therapies.